lformation of the nuclear envelope al models when FTase inhibitors Why is the 9w
ID: 151922 • Letter: L
Question
lformation of the nuclear envelope al models when FTase inhibitors Why is the 9wersed in animal models when 12. Which of the following is most likely to resu stunted growth and musculoskeletal degeneration A. Structurally supported nuclear envelopes B. Loss of the farnesyl group by prelamin A C. Methylation of prelamin A during post- are used? inhibitors catalyze the upstream PTase int of the modified farnesyl group. learin is made in the presence of FTase inhibitors. inhibitors are present. translational processing D. The improper manipulation of chromatin prelamin A is created when FTase during mitosis Frase inhibitors physically prevent progerin from locking to the nuclear rim. f the three amino acids found at the carboxyl D. 13. What changes occur in the DNA when a point mutation changes a cytosine to a thymine? A. One fewer hydrogen bond is formed between 10. One ho terminus of prelamin A could be A. tyrosine. B. tryptophan. C. phenylalanine. D. alanine. base pairs. B. A pyrimidine is changed into a purine. C. An-OH group is added to deoxyribose. D. The phosphate backbone is deformed due to base pair mismatch t. Which of the following is the most likely reason for the production of faulty prelamin A in individuals with progeria? A. The mutation introduces a new splice site, resulting in loss of RNA from one of the exons of prelamin A. B. The point mutation causes a stop codon to appear earlier in protein sequencing than is normal. C. The DNA splices and reforms at the lamin A gene, removing part of the protein's template strand D. The protein that is sequenced from prelamin A is less stable as a result of the mutation, and it denaturesExplanation / Answer
11. Option d is correct. 12.option A is correct . 14. Option A is correct. Prelamin A is present in nuclear envelope.
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