3. How is growth hormone (GH) release from the anterior pituitary regulated? Que
ID: 223904 • Letter: 3
Question
3. How is growth hormone (GH) release from the anterior pituitary regulated?
Question 3 options:
a. Stretch sensors in bone detect strain and stimulate the hypothalamus to increase release of GHRH, promoting bone growth.
b. GHRH and Somatostatin positively and negatively influence the level of a second messenger (cAMP) in GH releasing cells, respectively.
c. Elevated levels of GH cause reductions in circulating insulin and insulin-like growth factors, which feed back and stimulate GHRH production.
d. Unlike other anterior pituitary hormones that are controlled by the hypothalamus, the primary control for GH release comes from levels of secretin released by the duodenal endocrine cells.
e. Two of the above answers are correct.
6. Imagine that you've conducted an experiment on mice where regions of the hypothalamus were chronically stimulated via implanted electrodes. The result was that the stimulated population of animals exhibited increased metabolic rate, and that their liver cells had increased Na+ K+-ATPase activity relative to cells from implanted but not stimulated control animals. You'd reasonably conclude that the electrodes had
Question 6 options:
a. stimulated cells involved in the the release of vasopressin releasing hormone.
b. stimulated cells involved in the release of thyrotropin releasing hormone.
c. stimulated cells involved in the release of growth hormone releasing hormone.
d. stimulated cells involved in the release of aldosterone releasing hormone.
e. inhibited cells involved in the release of somatostatin releasing hormone.
a. Stretch sensors in bone detect strain and stimulate the hypothalamus to increase release of GHRH, promoting bone growth.
b. GHRH and Somatostatin positively and negatively influence the level of a second messenger (cAMP) in GH releasing cells, respectively.
c. Elevated levels of GH cause reductions in circulating insulin and insulin-like growth factors, which feed back and stimulate GHRH production.
d. Unlike other anterior pituitary hormones that are controlled by the hypothalamus, the primary control for GH release comes from levels of secretin released by the duodenal endocrine cells.
e. Two of the above answers are correct.
6. Imagine that you've conducted an experiment on mice where regions of the hypothalamus were chronically stimulated via implanted electrodes. The result was that the stimulated population of animals exhibited increased metabolic rate, and that their liver cells had increased Na+ K+-ATPase activity relative to cells from implanted but not stimulated control animals. You'd reasonably conclude that the electrodes had
Question 6 options:
a. stimulated cells involved in the the release of vasopressin releasing hormone.
b. stimulated cells involved in the release of thyrotropin releasing hormone.
c. stimulated cells involved in the release of growth hormone releasing hormone.
d. stimulated cells involved in the release of aldosterone releasing hormone.
e. inhibited cells involved in the release of somatostatin releasing hormone.
Explanation / Answer
3) The correct answer is option B- GHRH (growth hormone releasing hormone) and Somatostatin (Growth hormone inhibitng hormone) positively and negatively influence the level of a second messenger (cAMP) in GH releasing cells, respectively.
6) The correct option is B because the thyrotropin releasing hormone promotes TSH relaease which inturn causes the T3 and T4 release that upregulates the enzyme actuvity of Na+K+ATPase
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