Question Completion Status Why are pain signals transmitted by C-fibers slower,
ID: 281203 • Letter: Q
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Question Completion Status Why are pain signals transmitted by C-fibers slower, lingering, and harder to diminish with anesthetics? The diameter of the neurons is smaller O C-fibers are not myelinated C-fibers are less sensitive to lidocaine All of the above Q 10 points Save Answer Which is false regarding the function of general O They work by putting an individual into an unconscious state Their mechanism of action is unknown O Increased respiration can cause them to act faster OThey completely block action potentials in nociceptive neurons QUE 10 points How would you reverse a depolarizing blockade? O Activate ACHE O Inhibit AChE O Increase ACh levels O Activate AChR QUE 10 pointsExplanation / Answer
Why are pain signals transmitted by C-fibres slower,lingering and harder to diminish with anesthestics:
C fibres are small and unmyelinated,very slow in conducting,Respond to all type of noxious stimuli,Transmit prolonged dull pain and require high intensity stimuli to trigger a response.They are less sensitive to iodine.
Answer:All of the above.
Which one is false regarding the function of general anesthtics:
Increased respiration can cause them to act faster.
Pulmonary gas echange is disturbed during general anasthsia,both oxygenation and elimination of carbon dioxide is impaired.
How would you reverse a depolarizing blockade:
Inhibit AChE
Neuromuscular blocking drugs block neuromuscular transmission at the neuromuscular junction causing paralysis of affected skeletal muscles.This is accomplished by acting presynaptically via the inhibition of acetylcholine(ACh) synthesis or by acting post synaptically at the acetylcholine receptors of the motor nerve end plate.
Which is false regarding the functionality of barbitirates:
They have a stronger effect than benzodiazepines.
A class of drugs known as benzodiazepines has largely replaced barbiturates for both medical and recreational use.
What is the function of carbidopa
All of the above.
It is used to treat parkinson's disease.The combination of the two medicines carbidopa and levadopa primarily used to manage the symptoms of parkinson's disease.
Carbidopa prevents the breakdown of levadopa outside the brain.In the brain,the levadopa is broken down into dopamine by which it has its effect.
Levadopa can cross the blood brain barrier and it is a precursor to dopamine,the body can turn it into dopamine.Carbidopa helps levadopa cross BBB more effeciently.
Which f the following drug does not act by inhibiting GABAergic neurons that synapse onto DA neurons:
Cannabinoids
cannabis are used for its mental and physical effects such as high or stones feeling,a general change i perception and an increase in appetite.
Which of the following is the molecular mechanism of tolerance:
All of the above.
What neurotransmitter do drugs of abuse all modify:
GABA
If the neurotransmotter is inhibitory it will dampen the receiving neuron's electrical activity and reduce its likelihood of releasing the neurotransmitter.
How do opiate drugs exerts their effects on the CNS
They open Ca++ and close K+ channels.
What is the mechanism of action of nicotine
All of the above
Nicotine is the stimulant drug that acts as an agonist at nocotine acetylcholine receptors.These are ionotopic receptors composed up of five homomeric or heteromeric subunits.When nicotine enters the body,it is distributed quickly through the bloodstream and it can cross the blood brain barrier to enter the CNS.It acts on dopaminergic neurons.
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