d) Increased TRH, increased TSH 42. A subject is injected with TSH with TSH but
ID: 3512136 • Letter: D
Question
d) Increased TRH, increased TSH 42. A subject is injected with TSH with TSH but shows no uction. a) Hypothalamus gland b) Pituitary gland c) Thyroid gland d) None of the above 43. The semilunar pulmonary/aortic valves close at the a. Ventricular contraction b. Ventricular relaxation c.Atrial contraction d. Atrial relaxation 44. P-wave in ECG signifies atrial depolarization- b Ventricular depolarization c.Atrial repolarization d. Ventricular repolarization 45. A long P-R interval generally indicates a. Myocardial infraction b. Heart block 46. Sympathetic stimulation will cause a/anin a Increase b. Decrease 47. First heart sound is due to closing of the a) Semilunar valves b) Atrio-ventricular (AV) valves 48. During isovolumetric contraction, blood flows out of the ventricles. a) True b) False 49. Highest peak in the ECG waves (QRS) is due to depolarizaton. a Atrial b) Ventricular 50. T-wave in ECG is due to a) Atrial depolarization b) Ventricular depolarization c) Atrial repolarization d) Ventricular repolarizationExplanation / Answer
42) Thyroid gland.
TSH released from pituitary and TRH released from hypothalamus regulates the functioning of thyroid hormones.hence,after TSH administration if no response is generated than there is defect in the thyroid hormone.
Pituitary will be affected when there is no TSH hence no feedback inhibition.
43) b} ventricular diastole.
44) a} atrial depolarization
45) b} Heart block
Wenckebach phenomenon is seen.
46) a} Increase
47) b} Atrioventricular valve
48) b} False
Although contraction of ventricles occur,but there is no emptying in this phase.
49) b} ventricular depolarization
50) {d} ventricular repolarization
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