Complete the table for the following hormones. oxytocin GH Chemical make-up of h

Question

Complete the table for the following hormones.

oxytocin

GH

Chemical make-up of hormone: LS or NLS

Half-life

Carrier: yes/no; if yes, name the carrier

Feedback mechanism

Cell/ Tissue / Organ Source where it is produced

Cell / tissue / organ from where it is released

Stimulating Triggers

Inhibiting Triggers

Receptor Location

Membrane signaling mechanism

Overall impact on carb metabolism; glucose-sparing: yes or no

Overall impact on fat metabolism

Overall impact on protein metabolism

Overall impact on reproductive functions

Permissive effects

Non metabolic functions

oxytocin

GH

Chemical make-up of hormone: LS or NLS

Half-life

Carrier: yes/no; if yes, name the carrier

Feedback mechanism

Cell/ Tissue / Organ Source where it is produced

Cell / tissue / organ from where it is released

Stimulating Triggers

Inhibiting Triggers

Receptor Location

Membrane signaling mechanism

Overall impact on carb metabolism; glucose-sparing: yes or no

Overall impact on fat metabolism

Overall impact on protein metabolism

Overall impact on reproductive functions

Permissive effects

Non metabolic functions

Explanation / Answer

enhances glucose uptake by stimulation of intracellular release of calcium, and activation of phosphoinositid-3-kinase (PI3K), calcium-calmodulin kinase kinase (Ca-CAMKK), and AMP-activated protein kinase (AMPK).

induces fatty acid oxidation and lipolysis, and formation of small adipocytes.

induces insulin secretion via phosphoinositide (PI) turnover and activation of protein kinase C

Milk ejection reflex

Uterine contraction

sexual attraction

prostate growth

sex hormones

niacin

influencing social behavior and emotion.

romantic attachment

social behaviour

homeostasis

Stimulates the immune system

Increases muscle mass

Oxytocin GH Chemical make-up of hormone: LS or NLS neuropeptide peptide Half-life 3-5 minutes 10-20minutes Carrier: yes/no; if yes, name the carrier Growth hormone-binding protein (GHBP) Feedback mechanism positive negatiive Cell/ Tissue / Organ Source where it is produced paraventricular nucleus of the hypothalamus Cell / tissue / organ from where it is released posterior pituitary anterior lobe of the pituitary gland Stimulating Triggers child birth and breast feeding IGF-1. Inhibiting Triggers vasopressin inhibition insulin Receptor Location chromosome 3p25 liver, muscle and adipose tissue. Membrane signaling mechanism through G protein coupled receptors Growth hormone receptors Overall impact on carb metabolism; glucose-sparing: yes or no

enhances glucose uptake by stimulation of intracellular release of calcium, and activation of phosphoinositid-3-kinase (PI3K), calcium-calmodulin kinase kinase (Ca-CAMKK), and AMP-activated protein kinase (AMPK).

does not affect Overall impact on fat metabolism

induces fatty acid oxidation and lipolysis, and formation of small adipocytes.

increase in lipid oxidation) Overall impact on protein metabolism

induces insulin secretion via phosphoinositide (PI) turnover and activation of protein kinase C

protein anabolism Overall impact on reproductive functions

Milk ejection reflex

Uterine contraction

sexual attraction

prostate growth

Growth of body during uberty Permissive effects along with cortisol, estrogen and prolactin in milk production and ejection.

sex hormones

niacin

Non metabolic functions

influencing social behavior and emotion.

romantic attachment

social behaviour

homeostasis

Stimulates the immune system

Increases muscle mass

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