Augmentation in blood viscosity may be defined as the confrontation to natural f

Question

Augmentation in blood viscosity may be defined as the confrontation to natural flow, can also be picked out as a contributing factor in stress as well the debilitating atherosclerosis which is an influence upon the morbidity/mortality linked with tissue ischemia. It has been assessed how the effects of hematocrits upon rising viscosity, and potential associated changes in blood pressure, flow rate, as well as the corresponding physiologic reimbursement ratios.

The area where natural blood flow is affected the most by viscosity is the capillary bed, which has a smaller width than erythrocytes. Therefore, there is a chance of hypotension along with stasis in this location if vasodilation is implemented for escalating the blood flow rate. Accommodating blood strain amplification may comprise an additional circulatory work load with the added implication of hypertension, so as a result it provides superior outcomes in patients with atherosclerosis and hypertension as well as ischemia to improve blood flow percentage by diminishing viscosity. In a nutshell, a depreciated blood viscosity standard may attribute towards a minor blood pressure level, abrasion and injury on the vessel lumen. Along with this it may decrease the overall rate atherosclerotic process. Furthermore, supplementary research in this area shall most likely pave the way to auxiliary biologic and medical advantages.

Explanation / Answer

athero sclerosis ; it is a disease where the the clogging of arteries due to white blood cells where wall thickens . it is due to chronic inflammatory due to WBC cells . there will be wo types of plaques one is soft palques this is very dangerous , when this kind of palques   ruptures it causes the clot in the blood , so it leads to the morbidity or mortality with deperivation of the oxygen to the tissue leading to th ischemia , this leads to the infarction of the heart . the mechanism of atherosclerosis is the   the leukocytes begin to attack the endothelial cells of the artery , thios causes the inflamattion of the artery ,thre by the lesion lead to grow , this causes the abruption of the blood ,   ther will be marked increase in the endothelian 1 enzyme activity , this causes the obstruction of arteries

diagnosis ;

this causes the viscosity and velocity of the blood flowing in the artery . due to stress test we can recognise the disease . the most importanat in finding the nitirc oxide which functions between the blood vessel and cellwa the reduced NO plays a imporatant role in the pathophysiology of endothelial dysfunction , use of beta blockers and angotensin i inhibitors may reverse the condition , the use of acb inhibitors is very useful in diagnosis and prognosis of the disease . recognizing the acetyl choline relase is a mjor imortanat factor in relase of the disesase . to conclude the use of nitric oxide in cure of the disease , we need to do more research on the disease. nitric oxide plays much imortanat role in endothelial dysfuctioning and can be act as marker . which causes the vasodilation in the arteries

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