According to our discussion in lecture, based on understanding of HIV evolution

Question

According to our discussion in lecture, based on understanding of HIV evolution and the existence of trade-offs, why would someone with HIV take a break from using a particular drug to treat his/her HIV? To allow the body time to recover from the negative side-effects of the drug treatment To allow time for the population of virus that is susceptible to that drug to out-compete resistant virus Someone would only stop using a particular drug if they had been determined to be completely free of the virus What does the Gram stain do? It differentiates all antibiotic resistant bacteria by turning them dark purple as opposed to pink for antibiotic-susceptible bacteria Makes the vast majority of bacteria turn dark purple Differentiates among groups of bacteria by the structure of their cell wall According to our discussion in lecture, how do HIV drugs interfere with the "life" cycle of the HIV virus? By interfering with reverse transcriptase By interfering with virus assembly None of these Both of these

Explanation / Answer

ans-17: stoping Drug consumption to recover from prolonged toxicity of side effect is correct answer. HIV drugs cause sideeffects like decrease immunity, sostopping drug would make the patient recover from these types of sideeffects.

ans- 18: answer is (a), where gram stainin differetiates bacteria based on antibiotic resistance. Gram positive bacteria uptake the crystal violet stain because of their structure of walls and stains purple because their wall have thick peptidoglycans. While gram negative bacteria do not uptake crystal violet stain because of absence og thick wall and stains pink. these bacteria susseptible to antibiotic.

ans-19: answer is both of these, HIV drug is actually an analog (having similar structure but different function) of reverse transcriptase enzyme that inhibit viral genome replication by halting DNA synthesis from RNA. Another method of treatment is non reverse transcription enzyme strategy where this drug target viral protein so that no toxic viral proteins do get assembled.

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