3) If static loading doesn\'t promote osteogenesis, what doesthis mean for isome

Question

3) If static loading doesn't promote osteogenesis, what doesthis mean for isometric exercises?

4) What types of exercise and exercise schedule would yousuggest for healthy adults needing to increase bone mass?

5) What types of exercise and exercise schedule would yousuggest for effective osteogenic potential in children?

6) What is the relationship among running speed, bone strain,and number of loading cycles?

7) What was the hypothesis in the stress fracture study? Did thefindings support the hypothesis? Why or why not?

8) What were some assumptions the authors made in their stressfracture model? Do you agree with these assumptions? Why or why not?

9) What happened to peak tibial forces as speed increased? When did the probability of stress fractures peak?

10) As a coach/trainer/therapist, how might you use thisinformation to decrease the probability of stress fractures?

Explanation / Answer

Under 50–100 kPastatic pressure, proliferation was inhibited and apoptosis wasenhanced, but the cellular phenotype could be maintained. Highpressure (250–500 kPa) totally inhibited the bioactivityof the osteoblasts and induced necrosis. Mineralization nodulesdecreased significantly under 100 kPa pressure, while nonodules could be found under 250 and 500 kPapressureOsteogenic changes are seen in the tibia with the exposureof dynamic loading.

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