A 36-year-old male has been suffering with what appears to be upper gastric pain

Question

A 36-year-old male has been suffering with what appears to be upper gastric pain for approximately one month. One Sunday morning the pain is unbearable and he decides to go to the emergency room at the local hospital. He describes it as a rather sharp pain that last for 2 to 3 hours, and then it starts to resolve. He indicates that he has a vomited three times, and often suffers from nausea intermittently. He also says that these bouts do not necessarily occur during or after eating. And ultrasound promptly discovers several gallstones within the gallbladder and several small ones that appeared to be blocking his cystic duct.
What substance is readily responsible for gallbladder contraction? What initiating physiological event must proceed the secretion of the substanc?
What could possibly be a consequence of not having this situation medically corrected?
Does secretin have any role in this event?
Why is enterogastrone not likely to be involved in this scenari? A 36-year-old male has been suffering with what appears to be upper gastric pain for approximately one month. One Sunday morning the pain is unbearable and he decides to go to the emergency room at the local hospital. He describes it as a rather sharp pain that last for 2 to 3 hours, and then it starts to resolve. He indicates that he has a vomited three times, and often suffers from nausea intermittently. He also says that these bouts do not necessarily occur during or after eating. And ultrasound promptly discovers several gallstones within the gallbladder and several small ones that appeared to be blocking his cystic duct.
What substance is readily responsible for gallbladder contraction? What initiating physiological event must proceed the secretion of the substanc?
What could possibly be a consequence of not having this situation medically corrected?
Does secretin have any role in this event?
Why is enterogastrone not likely to be involved in this scenari?
What substance is readily responsible for gallbladder contraction? What initiating physiological event must proceed the secretion of the substanc?
What could possibly be a consequence of not having this situation medically corrected?
Does secretin have any role in this event?
Why is enterogastrone not likely to be involved in this scenari?

Explanation / Answer

Cholecystokinin (CCK) relaxes Sphincter of oddi. This is a cap present on the surface of gall bladder which does not allow bile to come out. When CCK relaxed Sphincter of oddi, then gall bladder relaxes and bile comes out. In the above condition normal release of CCK, relaxed the gall bladder------> bile released in cystic oddi------> along with bile gall stones also came in the cystic duct-----> blockage of the cystic duct.

If this situation is not corrected medically it will further lead to cholecystitis. This is an inflammation/ swelling of gall bladder. This condition is painful and may require surgery.

Duodenum mucosa secretes a number of hormones in the gastrointestinal area. Any of these hormones will be known as enterogastrone. Enterogastrone includes secretin, cholecystokinin and gastric inhibitory peptide.

Secretin enhances the effect of cholecystokinin, and increases it's work efficiency. Since the above problem is related with CCK, therefore secretin is also involved in further enhancing the problem.

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