Read the following article: Moore, A.S. & Shepard, L.H. (2014). Myasthenia gravi
ID: 87416 • Letter: R
Question
Read the following article:
Moore, A.S. & Shepard, L.H. (2014). Myasthenia gravis vs. guillain-barre syndrome what’s the difference? Nursing Made Incredibly Easy! 12 (4). 21-30.
Initial Discussion Post:
Select one (1) of the following Nursing Diagnoses:
Impaired Spontaneous Ventilation
Impaired Swallowing
Care Giver Role Strain
Address the following:
Is the nursing diagnoses that you selected appropriate for the patient with a diagnosis of myasthenia gravis, Guillain-Barre syndrome or both? Explain your answer.
Provide an outcome for the nursing diagnosis that you selected making sure that is specific to the needs of the patient with myasthenia gravis or Guillain-Barre syndrome.
Identify two (2) interventions that will help your patient with myasthenia gravis or Guillain-Barre syndrome reach the outcome for the nursing diagnosis.
Base your initial post on your readings and research of this topic. Your initial post must contain a minimum of 250 words. References, citations, and repeating the question do not count towards the 250 word minimum.
Explanation / Answer
The nursing diagnoses myasthenia gravis. Impaired Spontaneous Ventilation (or difficulty breathing), impaired swallowing and care giver role strain are the typical symptoms of myasthenia gravis. They are as a result of the weakness in the voluntary skeletal muscles.
The voluntary muscles of the body are controlled by nerve impulses arising in the brain. The nerve impulses travel down the nerves. These nerve fibers connect with muscle fibers though a space between the nerve ending and the muscle fiber. This space is called the neuromuscular junction. When the nerve impulse originating in the brain arrives at this nerve ending, it releases a acetylcholine which travels across the space to the muscle fiber side of the neuromuscular junction where it attaches to many receptor sites. The muscle contracts when enough of the receptor sites have been activated by the acetylcholine. In Myasthenia gravis, there is a decrease in the number of receptor sites that bind acetyl choline. There is an increase in the auto-antibodies that destroy the receptor sites and so the muscles become weak and the acetylcholine cannot activate enough receptor sites at the neuromuscular junction.
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