When 16:1 6 fatty acids are newly synthesized in the liver, they have two potent

Question

When 16:16 fatty acids are newly synthesized in the liver, they have two potential metabolic fates; one is to be processed for export into the circulation, the other is to be oxidized by beta-oxidation to generate acetyl-CoA for ATP production. Which of the following determines how newly generated 16:0 fatty acids will be handled?

Answers:

Increased gluconeogenesis will require acetyl-CoA from oxidation of fatty acids

Glucagon signaling will direct fatty acids into the oxidative pathway

Elevated concentration of malonyl-CoA

Elevated concentration of NADPH

Increased ketogenesis will require acetyl-CoA from oxidation of fatty acids

When 16:16 fatty acids are newly synthesized in the liver, they have two potential metabolic fates; one is to be processed for export into the circulation, the other is to be oxidized by beta-oxidation to generate acetyl-CoA for ATP production. Which of the following determines how newly generated 16:0 fatty acids will be handled?

Answers:

Increased gluconeogenesis will require acetyl-CoA from oxidation of fatty acids

Glucagon signaling will direct fatty acids into the oxidative pathway

Elevated concentration of malonyl-CoA

Elevated concentration of NADPH

Increased ketogenesis will require acetyl-CoA from oxidation of fatty acids

Explanation / Answer

The hormone Glucagon is catabolic hormone i.e. a hormone that leads to breaking down type of metabolism where substrates are utilized while the hormone Insulin is anabolic hormone. So increase in the levels of Glucagon will lead to oxidation of fatty acids, a form of catabolism. Hence the option: Glucagon signaling will direct fatty acid into the oxidative pathway is true.

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