At the molecular level what are the effects of nicotine on the nervous system? H
ID: 3518608 • Letter: A
Question
At the molecular level what are the effects of nicotine on the nervous system? How are these effects exerted? How are these effects similar to or different from those if curare? How do these effects explain some of the physiological consequences of smoking? At the molecular level what are the effects of nicotine on the nervous system? How are these effects exerted? How are these effects similar to or different from those if curare? How do these effects explain some of the physiological consequences of smoking?Explanation / Answer
Nicotine has an adverse effect on the body when it enters the body through smoking. The high breathing, increasing heart rate and blood pressure is highly increased due to the adrenaline secreation. This effect can cause a severe damage to the vital body organ of lungs, heart. There is an increase chance of heart attack, stokes and lungs diseases. Nicotine can constrict the blood vessels and thereby increases the blood flow and resulting increasing the blood pressure. It also increases the deposition of fat in the blood vessels.
During the smoking nicotine can causes neuro adaptation often resulting in tolerance and the effect of the nicotine on the central nervous system leads to the helps to overcome the distress. This can be explained by the decrease process of the sympathic nervous system with is associated wiith stress. This activity of the nicotine on stress is still under research process. So the actual process or the activity of the neuronal response on stress is not clear.
Curare represents an alkaloid arrow poison, which competes and reversibly inhibits the nAChR (or nicotinic acetylcholine receptor) at the motor end plate, leading to decrease in the value of end plate potential. Nicotine is able to activate the AChR at the neuromuscular junction, the physiological effects of smoking. Curare poison competitively and reversible inhibit nicotine acetylcholine receptor at neuromuscular junction.This might inhibit the depolarisation of motor end plate, which in turn might lead to stalling the transmission of signals at a neuromuscular synapse. It paralyses skeletal muscles and can ultimately lead to death due to asphyxiation by paralyzing Diaphragm. Overdose may be lethal because it leads to paralysis of Diaphragm and long time cessation of breathing can lead to cardiac failure.
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